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Effects of Swimming Exercise on Limbic and Motor Cortex Neurogenesis in the Kainate-Lesion Model of Temporal Lobe Epilepsy.

Cardiovascular psychiatry and neurology

Gorantla VR, Sirigiri A, Volkova YA, Millis RM.
PMID: 27313873
Cardiovasc Psychiatry Neurol. 2016;2016:3915767. doi: 10.1155/2016/3915767. Epub 2016 May 22.

Temporal lobe epilepsy (TLE) is a common neurological disease and antiseizure medication is often inadequate for preventing apoptotic cell death. Aerobic swimming exercise (EX) augments neurogenesis in rats when initiated immediately in the postictal period. This study tests the...

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Gorantla VR, Sirigiri A, Volkova YA, et al. Effects of Swimming Exercise on Limbic and Motor Cortex Neurogenesis in the Kainate-Lesion Model of Temporal Lobe Epilepsy. Cardiovasc Psychiatry Neurol. 2016;2016:3915767doi: 10.1155/2016/3915767.
Gorantla, V. R., Sirigiri, A., Volkova, Y. A., & Millis, R. M. (2016). Effects of Swimming Exercise on Limbic and Motor Cortex Neurogenesis in the Kainate-Lesion Model of Temporal Lobe Epilepsy. Cardiovascular psychiatry and neurology, 20163915767. https://doi.org/10.1155/2016/3915767
Gorantla, Vasavi R, et al. "Effects of Swimming Exercise on Limbic and Motor Cortex Neurogenesis in the Kainate-Lesion Model of Temporal Lobe Epilepsy." Cardiovascular psychiatry and neurology vol. 2016 (2016): 3915767. doi: https://doi.org/10.1155/2016/3915767
Gorantla VR, Sirigiri A, Volkova YA, Millis RM. Effects of Swimming Exercise on Limbic and Motor Cortex Neurogenesis in the Kainate-Lesion Model of Temporal Lobe Epilepsy. Cardiovasc Psychiatry Neurol. 2016;2016:3915767. doi: 10.1155/2016/3915767. Epub 2016 May 22. PMID: 27313873; PMCID: PMC4893441.
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Intercellular interactomics of human brain endothelial cells and th17 lymphocytes: a novel strategy for identifying therapeutic targets of CNS inflammation.

Cardiovascular psychiatry and neurology

Haqqani AS, Stanimirovic DB.
PMID: 21755032
Cardiovasc Psychiatry Neurol. 2011;2011:175364. doi: 10.1155/2011/175364. Epub 2011 Jun 13.

Leukocyte infiltration across an activated brain endothelium contributes to the neuroinflammation seen in many neurological disorders. Recent evidence shows that IL-17-producing T-lymphocytes (e.g., Th17 cells) possess brain-homing capability and contribute to the pathogenesis of multiple sclerosis and cerebral ischemia....

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Haqqani AS, Stanimirovic DB. Intercellular interactomics of human brain endothelial cells and th17 lymphocytes: a novel strategy for identifying therapeutic targets of CNS inflammation. Cardiovasc Psychiatry Neurol. 2011;2011:175364doi: 10.1155/2011/175364.
Haqqani, A. S., & Stanimirovic, D. B. (2011). Intercellular interactomics of human brain endothelial cells and th17 lymphocytes: a novel strategy for identifying therapeutic targets of CNS inflammation. Cardiovascular psychiatry and neurology, 2011175364. https://doi.org/10.1155/2011/175364
Haqqani, Arsalan S, and Stanimirovic, Danica B. "Intercellular interactomics of human brain endothelial cells and th17 lymphocytes: a novel strategy for identifying therapeutic targets of CNS inflammation." Cardiovascular psychiatry and neurology vol. 2011 (2011): 175364. doi: https://doi.org/10.1155/2011/175364
Haqqani AS, Stanimirovic DB. Intercellular interactomics of human brain endothelial cells and th17 lymphocytes: a novel strategy for identifying therapeutic targets of CNS inflammation. Cardiovasc Psychiatry Neurol. 2011;2011:175364. doi: 10.1155/2011/175364. Epub 2011 Jun 13. PMID: 21755032; PMCID: PMC3130966.
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Case series of post-thrombolysis patients undergoing hemicraniectomy for malignant anterior circulation ischaemic stroke.

Cardiovascular psychiatry and neurology

Williams A, Sittampalam M, Barua N, Mohd Nor A.
PMID: 21541210
Cardiovasc Psychiatry Neurol. 2011;2011:254569. doi: 10.1155/2011/254569. Epub 2011 Apr 18.

While ischaemic stroke remains a leading cause of death and disability, there have been recent advancements in treatment modalities including thrombolysis and decompressive hemicraniectomy. A retrospective review of patients treated in our NHS teaching hospital, in Plymouth (UK), over...

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Williams A, Sittampalam M, Barua N, et al. Case series of post-thrombolysis patients undergoing hemicraniectomy for malignant anterior circulation ischaemic stroke. Cardiovasc Psychiatry Neurol. 2011;2011:254569doi: 10.1155/2011/254569.
Williams, A., Sittampalam, M., Barua, N., & Mohd Nor, A. (2011). Case series of post-thrombolysis patients undergoing hemicraniectomy for malignant anterior circulation ischaemic stroke. Cardiovascular psychiatry and neurology, 2011254569. https://doi.org/10.1155/2011/254569
Williams, A, et al. "Case series of post-thrombolysis patients undergoing hemicraniectomy for malignant anterior circulation ischaemic stroke." Cardiovascular psychiatry and neurology vol. 2011 (2011): 254569. doi: https://doi.org/10.1155/2011/254569
Williams A, Sittampalam M, Barua N, Mohd Nor A. Case series of post-thrombolysis patients undergoing hemicraniectomy for malignant anterior circulation ischaemic stroke. Cardiovasc Psychiatry Neurol. 2011;2011:254569. doi: 10.1155/2011/254569. Epub 2011 Apr 18. PMID: 21541210; PMCID: PMC3085311.
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Role of Hypertension in Aggravating Abeta Neuropathology of AD Type and Tau-Mediated Motor Impairment.

Cardiovascular psychiatry and neurology

Díaz-Ruiz C, Wang J, Ksiezak-Reding H, Ho L, Qian X, Humala N, Thomas S, Martínez-Martín P, Pasinetti GM.
PMID: 19936102
Cardiovasc Psychiatry Neurol. 2009;2009:107286. doi: 10.1155/2009/107286. Epub 2009 Sep 17.

Epidemiological evidence suggests that hypertension may accelerate the onset and progression of Alzheimer's disease (AD). In this study, we explored the role of hypertension in the neurodegenerative changes associated with Abeta and tau aggregation. We induced hypertension in APP(swe)...

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Díaz-Ruiz C, Wang J, Ksiezak-Reding H, et al. Role of Hypertension in Aggravating Abeta Neuropathology of AD Type and Tau-Mediated Motor Impairment. Cardiovasc Psychiatry Neurol. 2009;2009:107286doi: 10.1155/2009/107286.
Díaz-Ruiz, C., Wang, J., Ksiezak-Reding, H., Ho, L., Qian, X., Humala, N., Thomas, S., Martínez-Martín, P., & Pasinetti, G. M. (2009). Role of Hypertension in Aggravating Abeta Neuropathology of AD Type and Tau-Mediated Motor Impairment. Cardiovascular psychiatry and neurology, 2009107286. https://doi.org/10.1155/2009/107286
Díaz-Ruiz, C, et al. "Role of Hypertension in Aggravating Abeta Neuropathology of AD Type and Tau-Mediated Motor Impairment." Cardiovascular psychiatry and neurology vol. 2009 (2009): 107286. doi: https://doi.org/10.1155/2009/107286
Díaz-Ruiz C, Wang J, Ksiezak-Reding H, Ho L, Qian X, Humala N, Thomas S, Martínez-Martín P, Pasinetti GM. Role of Hypertension in Aggravating Abeta Neuropathology of AD Type and Tau-Mediated Motor Impairment. Cardiovasc Psychiatry Neurol. 2009;2009:107286. doi: 10.1155/2009/107286. Epub 2009 Sep 17. PMID: 19936102; PMCID: PMC2775676.
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S100B Protein in the Nervous System and Cardiovascular Apparatus in Normal and Pathological Conditions.

Cardiovascular psychiatry and neurology

Donato R, Heizmann CW.
PMID: 21076683
Cardiovasc Psychiatry Neurol. 2010;2010:929712. doi: 10.1155/2010/929712. Epub 2010 Nov 10.

No abstract available.

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Donato R, Heizmann CW. S100B Protein in the Nervous System and Cardiovascular Apparatus in Normal and Pathological Conditions. Cardiovasc Psychiatry Neurol. 2010;2010:929712doi: 10.1155/2010/929712.
Donato, R., & Heizmann, C. W. (2010). S100B Protein in the Nervous System and Cardiovascular Apparatus in Normal and Pathological Conditions. Cardiovascular psychiatry and neurology, 2010929712. https://doi.org/10.1155/2010/929712
Donato, Rosario, and Heizmann, Claus W. "S100B Protein in the Nervous System and Cardiovascular Apparatus in Normal and Pathological Conditions." Cardiovascular psychiatry and neurology vol. 2010 (2010): 929712. doi: https://doi.org/10.1155/2010/929712
Donato R, Heizmann CW. S100B Protein in the Nervous System and Cardiovascular Apparatus in Normal and Pathological Conditions. Cardiovasc Psychiatry Neurol. 2010;2010:929712. doi: 10.1155/2010/929712. Epub 2010 Nov 10. PMID: 21076683; PMCID: PMC2977937.
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Heart and brain tissue banks for research on co-occurring cardiovascular and neurological/psychiatric disorders.

Cardiovascular psychiatry and neurology

Ikonomovic MD.
PMID: 20029622
Cardiovasc Psychiatry Neurol. 2009;2009:427840. doi: 10.1155/2009/427840. Epub 2009 Apr 15.

Epidemiological studies point to a strong and possibly causal association of psychiatric and neurological disorders with cardiovascular disease (CVD). Mechanistic links between these co-occurring illnesses are not well understood. Better insight into their relationship could help identify novel diagnostic...

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Ikonomovic MD. Heart and brain tissue banks for research on co-occurring cardiovascular and neurological/psychiatric disorders. Cardiovasc Psychiatry Neurol. 2009;2009:427840doi: 10.1155/2009/427840.
Ikonomovic, M. D. (2009). Heart and brain tissue banks for research on co-occurring cardiovascular and neurological/psychiatric disorders. Cardiovascular psychiatry and neurology, 2009427840. https://doi.org/10.1155/2009/427840
Ikonomovic, Milos D. "Heart and brain tissue banks for research on co-occurring cardiovascular and neurological/psychiatric disorders." Cardiovascular psychiatry and neurology vol. 2009 (2009): 427840. doi: https://doi.org/10.1155/2009/427840
Ikonomovic MD. Heart and brain tissue banks for research on co-occurring cardiovascular and neurological/psychiatric disorders. Cardiovasc Psychiatry Neurol. 2009;2009:427840. doi: 10.1155/2009/427840. Epub 2009 Apr 15. PMID: 20029622; PMCID: PMC2790182.
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Omega-3 Polyunsaturated Fatty Acids (n-3 PUFAs) in Cardiovascular Diseases (CVDs) and Depression: The Missing Link?.

Cardiovascular psychiatry and neurology

Chang JP, Chen YT, Su KP.
PMID: 20029628
Cardiovasc Psychiatry Neurol. 2009;2009:725310. doi: 10.1155/2009/725310. Epub 2009 Sep 27.

Background. Based on epidemiological data, clinical trials, and meta-analytic reviews, omega-3 polyunsaturated fatty acids (n-3 PUFAs) seem to be a biological link between depression and cardiovascular diseases (CVDs). Presentation. Involvement of n-3 PUFAs in depression and CVDs may be...

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Chang JP, Chen YT, Su KP. Omega-3 Polyunsaturated Fatty Acids (n-3 PUFAs) in Cardiovascular Diseases (CVDs) and Depression: The Missing Link?. Cardiovasc Psychiatry Neurol. 2009;2009:725310doi: 10.1155/2009/725310.
Chang, J. P., Chen, Y. T., & Su, K. P. (2009). Omega-3 Polyunsaturated Fatty Acids (n-3 PUFAs) in Cardiovascular Diseases (CVDs) and Depression: The Missing Link?. Cardiovascular psychiatry and neurology, 2009725310. https://doi.org/10.1155/2009/725310
Chang, Jane Pei-Chen, et al. "Omega-3 Polyunsaturated Fatty Acids (n-3 PUFAs) in Cardiovascular Diseases (CVDs) and Depression: The Missing Link?." Cardiovascular psychiatry and neurology vol. 2009 (2009): 725310. doi: https://doi.org/10.1155/2009/725310
Chang JP, Chen YT, Su KP. Omega-3 Polyunsaturated Fatty Acids (n-3 PUFAs) in Cardiovascular Diseases (CVDs) and Depression: The Missing Link?. Cardiovasc Psychiatry Neurol. 2009;2009:725310. doi: 10.1155/2009/725310. Epub 2009 Sep 27. PMID: 20029628; PMCID: PMC2791235.
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The heart-brain connection begets cardiovascular psychiatry and neurology.

Cardiovascular psychiatry and neurology

Manev H.
PMID: 20029675
Cardiovasc Psychiatry Neurol. 2009;2009:546737. doi: 10.1155/2009/546737. Epub 2009 Jan 28.

No abstract available.

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Manev H. The heart-brain connection begets cardiovascular psychiatry and neurology. Cardiovasc Psychiatry Neurol. 2009;2009:546737doi: 10.1155/2009/546737.
Manev, H. (2009). The heart-brain connection begets cardiovascular psychiatry and neurology. Cardiovascular psychiatry and neurology, 2009546737. https://doi.org/10.1155/2009/546737
Manev, Hari. "The heart-brain connection begets cardiovascular psychiatry and neurology." Cardiovascular psychiatry and neurology vol. 2009 (2009): 546737. doi: https://doi.org/10.1155/2009/546737
Manev H. The heart-brain connection begets cardiovascular psychiatry and neurology. Cardiovasc Psychiatry Neurol. 2009;2009:546737. doi: 10.1155/2009/546737. Epub 2009 Jan 28. PMID: 20029675; PMCID: PMC2796392.
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Hypotheses on mechanisms linking cardiovascular and psychiatric/neurological disorders.

Cardiovascular psychiatry and neurology

Manev H.
PMID: 20029617
Cardiovasc Psychiatry Neurol. 2009;2009:197132. doi: 10.1155/2009/197132. Epub 2009 Oct 26.

No abstract available.

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Manev H. Hypotheses on mechanisms linking cardiovascular and psychiatric/neurological disorders. Cardiovasc Psychiatry Neurol. 2009;2009:197132doi: 10.1155/2009/197132.
Manev, H. (2009). Hypotheses on mechanisms linking cardiovascular and psychiatric/neurological disorders. Cardiovascular psychiatry and neurology, 2009197132. https://doi.org/10.1155/2009/197132
Manev, Hari. "Hypotheses on mechanisms linking cardiovascular and psychiatric/neurological disorders." Cardiovascular psychiatry and neurology vol. 2009 (2009): 197132. doi: https://doi.org/10.1155/2009/197132
Manev H. Hypotheses on mechanisms linking cardiovascular and psychiatric/neurological disorders. Cardiovasc Psychiatry Neurol. 2009;2009:197132. doi: 10.1155/2009/197132. Epub 2009 Oct 26. PMID: 20029617; PMCID: PMC2790123.
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Are cardiovascular risk factors associated with verbal learning and memory impairment in patients with schizophrenia? A cross-sectional study.

Cardiovascular psychiatry and neurology

Lancon C, Dassa D, Fernandez J, Richieri R, Padovani R, Boyer L.
PMID: 23227310
Cardiovasc Psychiatry Neurol. 2012;2012:204043. doi: 10.1155/2012/204043. Epub 2012 Nov 19.

Objective. The aim of this study is to assess the relationships of cardiovascular risk factors with verbal learning and memory in patients with schizophrenia. Methods and Design. cross-sectional study. Inclusion Criteria. Diagnosis of schizophrenia according to the DSM-IV-TR criteria....

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Lancon C, Dassa D, Fernandez J, et al. Are cardiovascular risk factors associated with verbal learning and memory impairment in patients with schizophrenia? A cross-sectional study. Cardiovasc Psychiatry Neurol. 2012;2012:204043doi: 10.1155/2012/204043.
Lancon, C., Dassa, D., Fernandez, J., Richieri, R., Padovani, R., & Boyer, L. (2012). Are cardiovascular risk factors associated with verbal learning and memory impairment in patients with schizophrenia? A cross-sectional study. Cardiovascular psychiatry and neurology, 2012204043. https://doi.org/10.1155/2012/204043
Lancon, Christophe, et al. "Are cardiovascular risk factors associated with verbal learning and memory impairment in patients with schizophrenia? A cross-sectional study." Cardiovascular psychiatry and neurology vol. 2012 (2012): 204043. doi: https://doi.org/10.1155/2012/204043
Lancon C, Dassa D, Fernandez J, Richieri R, Padovani R, Boyer L. Are cardiovascular risk factors associated with verbal learning and memory impairment in patients with schizophrenia? A cross-sectional study. Cardiovasc Psychiatry Neurol. 2012;2012:204043. doi: 10.1155/2012/204043. Epub 2012 Nov 19. PMID: 23227310; PMCID: PMC3512211.
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Ways of coping and biomarkers of an increased atherothrombotic cardiovascular disease risk in elderly individuals.

Cardiovascular psychiatry and neurology

von Känel R, Mausbach BT, Dimsdale JE, Mills PJ, Patterson TL, Ancoli-Israel S, Ziegler MG, Roepke SK, Allison M, Grant I.
PMID: 22848795
Cardiovasc Psychiatry Neurol. 2012;2012:875876. doi: 10.1155/2012/875876. Epub 2012 Jul 17.

Objective. To investigate the relationship between coping and atherothrombotic biomarkers of an increased cardiovascular disease (CVD) risk in the elderly. Methods. We studied 136 elderly caregiving and noncaregiving men and women who completed the Ways of Coping Checklist to...

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von Känel R, Mausbach BT, Dimsdale JE, et al. Ways of coping and biomarkers of an increased atherothrombotic cardiovascular disease risk in elderly individuals. Cardiovasc Psychiatry Neurol. 2012;2012:875876doi: 10.1155/2012/875876.
von Känel, R., Mausbach, B. T., Dimsdale, J. E., Mills, P. J., Patterson, T. L., Ancoli-Israel, S., Ziegler, M. G., Roepke, S. K., Allison, M., & Grant, I. (2012). Ways of coping and biomarkers of an increased atherothrombotic cardiovascular disease risk in elderly individuals. Cardiovascular psychiatry and neurology, 2012875876. https://doi.org/10.1155/2012/875876
von Känel, Roland, et al. "Ways of coping and biomarkers of an increased atherothrombotic cardiovascular disease risk in elderly individuals." Cardiovascular psychiatry and neurology vol. 2012 (2012): 875876. doi: https://doi.org/10.1155/2012/875876
von Känel R, Mausbach BT, Dimsdale JE, Mills PJ, Patterson TL, Ancoli-Israel S, Ziegler MG, Roepke SK, Allison M, Grant I. Ways of coping and biomarkers of an increased atherothrombotic cardiovascular disease risk in elderly individuals. Cardiovasc Psychiatry Neurol. 2012;2012:875876. doi: 10.1155/2012/875876. Epub 2012 Jul 17. PMID: 22848795; PMCID: PMC3405559.
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The S100B/RAGE Axis in Alzheimer's Disease.

Cardiovascular psychiatry and neurology

Leclerc E, Sturchler E, Vetter SW.
PMID: 20672051
Cardiovasc Psychiatry Neurol. 2010;2010:539581. doi: 10.1155/2010/539581. Epub 2010 Jun 21.

Increasing evidence suggests that the small EF-hand calcium-binding protein S100B plays an important role in Alzheimer's disease. Among other evidences are the increased levels of both S100B and its receptor, the Receptor for Advanced Glycation Endproducts (RAGEs) in the...

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Leclerc E, Sturchler E, Vetter SW. The S100B/RAGE Axis in Alzheimer's Disease. Cardiovasc Psychiatry Neurol. 2010;2010:539581doi: 10.1155/2010/539581.
Leclerc, E., Sturchler, E., & Vetter, S. W. (2010). The S100B/RAGE Axis in Alzheimer's Disease. Cardiovascular psychiatry and neurology, 2010539581. https://doi.org/10.1155/2010/539581
Leclerc, Estelle, et al. "The S100B/RAGE Axis in Alzheimer's Disease." Cardiovascular psychiatry and neurology vol. 2010 (2010): 539581. doi: https://doi.org/10.1155/2010/539581
Leclerc E, Sturchler E, Vetter SW. The S100B/RAGE Axis in Alzheimer's Disease. Cardiovasc Psychiatry Neurol. 2010;2010:539581. doi: 10.1155/2010/539581. Epub 2010 Jun 21. PMID: 20672051; PMCID: PMC2905692.
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Showing 1 to 12 of 105 entries